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Metabolic regulation of neuronal plasticity by the energy sensor AMPK
Wyatt B. Potter
,
Kenneth J. O'Riordan
, David Barnett
, Susan M.K. Osting
, Matthew Wagoner
, Corinna Burger
, Avtar Roopra
University of Wisconsin-Madison
Research output
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peer-review
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Medicine and Dentistry
Nerve Cell Plasticity
100%
Metabolic Regulation
100%
Hydroxymethylglutaryl Coenzyme A Reductase Kinase
100%
Long-Term Potentiation
88%
Mechanistic Target of Rapamycin
66%
Energy Metabolism
55%
Diseases
11%
Brain Derived Neurotrophic Factor
11%
Homeostasis
11%
Epileptic Seizure
11%
Glycon
11%
Maturity Onset Diabetes of the Young
11%
Adenosine Triphosphate
11%
Cell Energy
11%
Promoter Region
11%
Translation (Protein Synthesis)
11%
Adenosine Phosphate
11%
Intractable Epilepsy
11%
Vidarabine
11%
5 Amino 4 Imidazolecarboxamide Riboside
11%
Epileptogenesis
11%
Kindling
11%
Kindling Model
11%
Hippocampal Slice
11%
RNA Transcription
11%
Neuroscience
Neuronal Plasticity
100%
AMP-activated Protein Kinase
100%
Long-Term Potentiation
88%
Mammalian Target of Rapamycin
66%
Energy Metabolism
55%
2-Deoxy-D-Glucose
22%
Brain-Derived Neurotrophic Factor
11%
Promoter Region
11%
Adenosine Triphosphate
11%
Cell Energy
11%
Metformin
11%
Intractable Epilepsy
11%
Adenosine Monophosphate
11%
Vidarabine
11%
Kindling Model
11%
Epileptogenesis
11%
5 Amino 4 Imidazolecarboxamide Riboside
11%
RNA Transcription
11%
Kindling
11%