Presenilins are novel substrates for TRAF6-mediated ubiquitination

Justin Mccarthy

doi:10.1016/j.cellsig.2013.05.015

Presenilins are novel substrates for TRAF6-mediated ubiquitination

Justin Mccarthy

Research output: Contribution to journal › Article › peer-review

Original language	English
Journal	Cellular Signalling
DOIs	https://doi.org/10.1016/j.cellsig.2013.05.015
Publication status	Published - 2013

UN SDGs

This output contributes to the following UN Sustainable Development Goals (SDGs)

SDG 3 Good Health and Well-being

Keywords

Presenilin
Ubiquitin
Ubiquitin ligase
Cell biology
Amyloid precursor protein secretase
Biology
Amyloid precursor protein
Transcription factor
Chemistry
Biochemistry
Alzheimer's disease
Gene
Internal medicine
Medicine
Disease

Access to Document

10.1016/j.cellsig.2013.05.015

https://openalex.org/W2056169537Licence: CC BY-NC-ND

Cite this

@article{50b21ac09f5c4c5b95bbcff37223808b,

title = "Presenilins are novel substrates for TRAF6-mediated ubiquitination",

keywords = "Presenilin, Ubiquitin, Ubiquitin ligase, Cell biology, Amyloid precursor protein secretase, Biology, Amyloid precursor protein, Transcription factor, Chemistry, Biochemistry, Alzheimer's disease, Gene, Internal medicine, Medicine, Disease",

author = "Justin Mccarthy",

year = "2013",

doi = "10.1016/j.cellsig.2013.05.015",

language = "English",

journal = "Cellular Signalling",

issn = "0898-6568",

publisher = "Elsevier Inc.",

}

TY - JOUR

T1 - Presenilins are novel substrates for TRAF6-mediated ubiquitination

AU - Mccarthy, Justin

PY - 2013

Y1 - 2013

KW - Presenilin

KW - Ubiquitin

KW - Ubiquitin ligase

KW - Cell biology

KW - Amyloid precursor protein secretase

KW - Biology

KW - Amyloid precursor protein

KW - Transcription factor

KW - Chemistry

KW - Biochemistry

KW - Alzheimer's disease

KW - Gene

KW - Internal medicine

KW - Medicine

KW - Disease

U2 - 10.1016/j.cellsig.2013.05.015

DO - 10.1016/j.cellsig.2013.05.015

M3 - Article

SN - 0898-6568

JO - Cellular Signalling

JF - Cellular Signalling

ER -