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Retinoid receptor signaling and autophagy in acute promyelocytic leukemia
Nina Orfali
,
Sharon L. McKenna
, Mary R. Cahill
, Lorraine J. Gudas
, Nigel P. Mongan
University College Cork
Cornell University
University of Nottingham
Research output
:
Contribution to journal
›
Review article
›
peer-review
Overview
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Medicine and Dentistry
Receptor
100%
Autophagy
100%
Acute Promyelocytic Leukemia
100%
Retinoid
100%
Tretinoin
42%
Leukemia Cell
28%
Chimeric Protein
28%
Arsenic Trioxide
28%
Retinol
14%
Signaling Molecule
14%
Cell Differentiation
14%
Cancer
14%
Hematopoiesis
14%
Carcinogenesis
14%
Chromosome Translocation
14%
Differentiation Therapy
14%
Leukemogenesis
14%
Protein Degradation
14%
Biochemistry, Genetics and Molecular Biology
Autophagy
100%
Retinoid
100%
Retinoid Receptor
100%
Tretinoin
60%
Chimeric Protein
40%
Arsenite
40%
Arsenous Acid
40%
Arsenic Trioxide
40%
Cellular Differentiation
20%
Hematopoiesis
20%
Carcinogenesis
20%
Protein Degradation
20%
Retinoic Acid
20%
Chromosomal Translocation
20%
Pharmacology, Toxicology and Pharmaceutical Science
Receptor
100%
Promyelocytic Leukemia
100%
Retinoid
100%
Retinoic Acid
42%
Chimeric Protein
28%
Arsenous Acid
28%
Arsenite
28%
Carcinogenesis
14%
Remission
14%
Elimination
14%
Retinol
14%
Chromosome Translocation
14%
Leukemogenesis
14%